What does ratchet slang mean?
What does ratchet slang mean?
Ratchet is a slang term in hip hop that, in its original sense, referred to an uncouth woman, and may be a Louisianan regiolect version of the word “wretched” or a variation of the word “ratshit.” The term has since been extended to have broader meanings and connotations and is no longer strictly bound by race or …
What is a synonym for Bougie?
Used to describe middle-class values in their attempt to give the semblance of discerning taste. bourgie. chichi. flamboyant.
What is a French Bougie?
Traditionally, the bougie (pronounced Boo-gee) is a measuring device developed in France that is in the form of a long, flexible tube and used in gastric sleeve surgery to guide surgeons when dividing the stomach. The tube comes in various sizes, and is measured in units known as a French (1 French or F=0.333 mm).
What do you need for intubation?
Equipment includes the following:
- Laryngoscope (see image below): Confirm that light source is functional prior to intubation.
- Laryngoscope handle, No.
- Endotracheal (ET) tube.
- Stylet.
- Syringe, 10 mL (to inflate ET tube balloon)
- Suction catheter (eg, Yankauer)
- Carbon dioxide detector (eg, Easycap)
- Oral and nasal airways.
What drugs are needed for intubation?
Common sedative agents used during rapid sequence intubation include etomidate, ketamine, and propofol. Commonly used neuromuscular blocking agents are succinylcholine and rocuronium.
Are you awake when you are intubated?
The more cooperative your patient, the more you can rely on local; perfectly cooperative patients can be intubated awake without any sedation at all. More commonly in the ED, patients will require sedation.
Why is atropine given before intubation?
Like fentanyl, it can be given before induction agents to facilitate endotracheal intubation. Atropine occasionally is used as a premedication. Its anticholinergic effects reduce ACH-mediated bradycardia that can accompany endotracheal intubation.
Why are paralytics used?
NMBAs are used in the ICU to improve patient-ventilator synchrony, enhance gas exchange, and diminish the risk of barotrauma. The most common reason for NMBA administration is to facilitate mechanical ventilatory support.
Why are paralytics used for intubation?
improves intubating conditions. makes ventilation easier. prevents the patient from interfering with peri-intubation procedures should sedation wear off. allowing the patient to wake is virtually never an option in the critically ill patient requiring intubation (proceed to surgical airway in the CICV situation)
Why are muscle relaxants used for intubation?
(1). Muscle relaxants are frequently used to facilitate endotracheal intubation during anesthesia induction. However, the administration of short-acting depolarizing muscle relaxants is closely related to postoperative myalgias, malignant hyperthermia, hyperkalemia, and increased intracranial or intraocular pressure.
Is sedation required for intubation?
Sedation and analgesia for intubation Laryngoscopy and intubation are uncomfortable; in conscious patients, a short-acting IV drug with sedative or combined sedative and analgesic properties is mandatory.
Can you intubate without a paralytic?
Facilitated intubation, also known as medication-facilitated intubation or sedation-facilitated intubation, refers to intubation performed using a sedative or anesthetic drug as an induction agent, without the use of a paralytic (neuromuscular blocking agent).
What is the difference between rocuronium and vecuronium?
Rocuronium had a faster onset time than vecuronium, but had a similar duration of action. Vecuronium had no significant cardiovascular effects. Rocuronium caused a rise in mean arterial pressure (10-15%) and a slight rise in heart rate (5-10%).
What is the reversal agent for propofol?
Unlike other sedation agents (e.g., midazolam, morphine), there is no reversal agent for propofol. Adverse effects must be treated until the drug is metabolized.
What is vecuronium used for?
Vecuronium bromide is an FDA approved peripherally acting, monoquarternary, steroidal, non-depolarizing neuromuscular blocker with an intermediate duration of action used during general anesthesia to facilitate endotracheal intubation, to aid in surgical relaxation, and, less commonly, in the intensive care setting to …
What is rocuronium and vecuronium?
Vecuronium and rocuronium are commonly used during induction of anesthesia to facilitate tracheal intubation, and mivacurium is frequently administered for maintenance of neuromuscular blockade to facilitate spontaneous recovery of neuromuscular function.
Is rocuronium a paralytic?
Rocuronium is a nondepolarizing paralytic agent that induces muscle paralysis by competitive antagonism at the acetyl-cholinergic receptor. Dosing of rocuronium can vary from 0.6—1.2 mg/kg. The onset of action is dose-dependent from 45—120 seconds, with a duration of action 30—90 minutes.
What is the antidote for rocuronium?
Sugammadex (ORG 25969) is a unique neuromuscular reversal drug; a novel cyclodextrin, the first in a new class of selective relaxant binding agents, which reverse neuromuscular blockade (NMB) with the aminosteroid non-depolarizing muscle relaxants rocuronium and vecuronium. Sugammadex can reverse moderate or deep NMB.
Is rocuronium long acting?
Rocuronium is an intermediate-acting nondepolarizing neuromuscular blocker with ED95 of 0.3 mg/kg. At a dosing range of 0.6 to 1.2 mg/kg, intubating conditions can be reached in 1 to 2 min with effects lasting until 20 to 35 min.
What is another name for rocuronium?
Introduced in 1994, rocuronium has rapid onset, and intermediate duration of action. It is commonly marketed under the trade names Zemuron and Esmeron.
How fast do you push rocuronium?
The recommended initial dose of rocuronium bromide, regardless of anesthetic technique, is 0.6 mg/kg. Neuromuscular block sufficient for intubation (80% block or greater) is attained in a median (range) time of 1 (0.4 to 6) minute(s) and most patients have intubation completed within 2 minutes.
How is rocuronium cleared?
Rocuronium, like vecuronium, is eliminated primarily through hepatobiliary excretion with < 1% metabolism. Since only 10% is eliminated through the kidneys,206 it is even less dependent on renal elimination than vecuronium.